9. True: A bruise occurs when blood leaks out of damaged vessels and into tissues.
10. True: Lactic acid in blood is released from muscle cells undergoing cellular respiration to produce the ATP needed for cell work.
11. True: Macrophages eliminate foreign bacteria by engulfing them using a process called phagocytosis then digesting them using enzymes found in lysosomes.
12. False: The blood cell stem cell which can develop into any of the formed elements is called a hematopoietic stem cell.
13. True: The amount of blood in an adult human varies depending on body size, but on average it is about five liters.
14. False: Anemia is a condition in which there are not enough healthy red blood cells to carry oxygen to the body's tissues. It is not contagious, and there are treatments and cures available.
15. True: The hormone erythropoietin is released from the kidneys in response to low blood oxygen levels, and its physiological effect is to stimulate red blood cell production in red marrow.
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The muscles that carry out contraction in the patellar reflex is
the group of hamstrings.
TRUE OR FALSE
Answer:
False. The muscles that carry out contraction in the patellar reflex are the quadriceps muscles.
Answer:
False
Explanation:
OB type questions:
1. What are the signs and symptoms of magnesium toxicity? What is the antidote?
2. What are the signs and symptoms for severe preeclampsia?
3. What medications are used for preterm labor?
4. What therapeutic procedures are used to prolong pregnancy?
1. Flushing or warmth of the skin, Nausea and vomiting. The antidote for magnesium toxicity is calcium gluconate or calcium chloride
2. High blood pressure and Protein in the urine.
3. Bed rest and Tocolytics
4. Cerclage, Cervical pessary,
What more should you know about magnesium toxicity?1. Signs and symptoms of magnesium toxicity, also known as hypermagnesemia, include: Flushing or warmth of the skin, Nausea and vomiting, Low blood pressure and Slow or irregular heartbeat, coma
2. Signs and symptoms of severe preeclampsia may include,
High blood pressure Protein in the urineSwelling, especially in the hands, feet, and face Headaches Vision problems Severe nausea and vomiting3. The most commonly used medications for preterm labor are:
Bed restTocolytics (medications that stop contractions)CorticoSteroid (to help the baby's lungs mature)4. Therapeutic procedures that can be used to prolong pregnancy in certain situations include: Cerclage, Cervical pessary, Bed rest, Prenatal corticosteroids
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Indirect costs of a high burden of chronic diseases among the working age population include
A. Pension costs
B. Health care costs
C. Social Security cost
D. Pension costs and Social security costs
Indirect costs of a high burden of chronic diseases among the working age population include pension costs, health care costs, social security cost, as well as loss of productivity, and reduced economic growth, resulting in a reduced quality of life.
Indirect costs of a high burden of chronic diseases among the working age population include various costs that are not directly linked to the disease itself. Indirect costs refer to the costs that are related to the treatment of diseases and are not directly related to the disease itself.
The costs are a result of the loss of productivity, reduced economic growth, and the impact on quality of life. These indirect costs are often overlooked and can have a significant impact on the economic stability of individuals and society as a whole.The loss of productivity can be attributed to missed days at work or reduced work capacity. The reduced economic growth is a result of decreased spending and lower taxes paid.
The cost of treatment and medication can also be a significant financial burden on families and individuals. Furthermore, indirect costs include reduced quality of life and increased poverty among the working-age population. Indirect costs of chronic disease are a significant burden on the economy and are often not fully accounted for when assessing the economic impact of chronic disease.
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When typing directions for pediatric medications, which of the following are examples of how to write the dose?
a)
Give 1 teaspoon (5 mL) three times a day.
b)
Give 1 tablespoon three times a day.
c)
Give 5 mL three times a day.
d)
Give 1 teaspoon three times a day.
When typing directions for pediatric medications, "give 1 teaspoon (5 mL) three times a day" and "give 5 mL three times a day" are two examples of how to write the dose.
The pediatric medication dosage is generally written in terms of milliliters (mL), and it's important to know the exact milliliter amount of the medication being given. Because children come in various shapes and sizes, the dose of medication is generally determined by weight or age.
It is recommended that medication be administered in the precise amounts directed by a doctor or pediatrician. A typical pediatric dosage for many medications is determined based on the child's age and weight. In general, pediatric medications are administered in milliliters (mL). Thus, "Give 1 teaspoon (5 mL) three times a day" and "Give 5 mL three times a day" are two examples of how to write the dose.
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Prion diseases or transmissible spongiform encephalopathies
(TSEs) are a family of rare progressive neurodegenerative disorders
caused by abnormal splicing of nucleotides.
True or flase
The correct answer is false
Prion diseases or transmissible spongiform encephalopathies (TSEs) are a family of rare progressive neurodegenerative disorders caused by the misfolding of normal cellular prion proteins. These misfolded proteins, called prions, accumulate in the brain and disrupt normal brain function, leading to the characteristic symptoms of TSEs. The abnormal splicing of nucleotides is not the cause of prion diseases.
The abnormal splicing of nucleotides is not the cause of prion diseases. Instead, it is the misfolding of the prion protein itself that triggers the pathogenesis of these diseases. The misfolded prions can induce the normal prion proteins to adopt the abnormal conformation, perpetuating the disease process.
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The National Quality Standard (NQS) sets the benchmark
for services across Australia. Identify and describe the following
three (3) quality areas that are most applicable to developing
cultural compet
Quality Area 1: Educational Program and Practice, Quality Area 6: Collaborative Partnerships with Families and Communities, and Quality Area 7: Governance and Leadership are the most applicable NQS quality areas for developing cultural competence.
Quality Area 1: Educational Program and Practice: This quality area focuses on promoting inclusive and culturally responsive educational programs.
It emphasizes the need for services to develop curriculum plans that respect and celebrate the diverse cultures and backgrounds of children and their families.
It involves incorporating culturally relevant resources, activities, and experiences to support children's learning and understanding of different cultures.
Quality Area 6: Collaborative Partnerships with Families and Communities: This quality area highlights the importance of building strong relationships with families and engaging with the local community.
It encourages services to actively involve families and communities in decision-making processes, seeking their input and valuing their cultural perspectives.
Effective collaboration helps services gain insights into the cultural practices, beliefs, and values of families, enabling them to tailor their approach to better support cultural diversity.
Quality Area 7: Governance and Leadership: This quality area focuses on the role of leadership and governance in promoting cultural competence.
It emphasizes the need for service leaders and management to demonstrate a commitment to diversity, inclusivity, and cultural responsiveness.
Effective governance and leadership provide a framework for developing and implementing policies, procedures, and strategies that support cultural competence across all aspects of service provision.
These three quality areas of the NQS provide a comprehensive framework for services to develop cultural competence by promoting inclusive educational programs, building collaborative partnerships.
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Please describe what your coping mechanisms (give and example of
when you would use it) are and what things could you work on for
doing self care? Is self care important? How can you help a patient
ad
Coping mechanisms are a range of strategies that people use to cope with difficult life situations. Coping mechanisms can be adaptive or maladaptive. People can develop maladaptive coping mechanisms to help them deal with stress. For example, someone who uses alcohol or drugs to cope with stress is using a maladaptive coping mechanism.
In my experience, whenever I encounter stress or anxiety, I try to cope by taking a break from whatever it is that's causing the stress. I find it helpful to take a walk outside or listen to some calming music. I also find it helpful to talk to someone about what's going on and how I'm feeling.
Self-care is important because it helps people maintain their physical, mental, and emotional health. It's important to prioritize self-care in order to maintain good mental health, which in turn helps to maintain good physical health. Self-care can include things like exercise, meditation, spending time with friends and family, and engaging in hobbies and other activities that bring you joy.
To help a patient with self-care, you can start by encouraging them to make time for themselves and prioritize self-care. You can also provide them with resources and information about self-care, such as exercise programs, support groups, and mindfulness techniques. You can also encourage them to seek professional help if they are struggling with mental health issues.
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A patient on your unit is found to have pulmonary tuberculosis (TB). What is the most appropriate precaution for the staff to take to prevent transmission of TB? 0 A Standard and contact precautions
B. Standard and airborne precautions O C. Standard precautions only O D. Standard and droplet precautions
B. Standard and airborne precautions.
It is crucial to adhere to the appropriate precautions to protect healthcare workers and prevent the spread of tuberculosis within the healthcare setting.
The most appropriate precaution for the staff to take to prevent transmission of pulmonary tuberculosis (TB) is Standard and airborne precautions. Tuberculosis is primarily spread through the air when an infected individual coughs, sneezes, or talks, releasing infectious droplets containing Mycobacterium tuberculosis (the bacteria that causes TB) into the air.
Standard precautions are the basic infection prevention practices that should be followed in all healthcare settings for all patients, regardless of their diagnosis. These precautions include hand hygiene, the use of personal protective equipment (e.g., gloves, masks, gowns), safe injection practices, and proper handling and disposal of contaminated materials.
In the case of tuberculosis, airborne precautions should be added to standard precautions because the infectious particles can remain suspended in the air for an extended period. Airborne precautions involve the use of specialized respiratory protection, such as N95 respirators or similar high-filtration masks, to protect healthcare workers from inhaling the infectious droplets. Additionally, patients with TB should be placed in a negative pressure isolation room, which helps prevent the spread of airborne particles to other areas of the healthcare facility.
Droplet precautions (option D) are not sufficient for preventing the transmission of TB because the infectious particles are smaller and can remain suspended in the air for longer distances. Droplet precautions are primarily used for diseases transmitted through larger respiratory droplets that travel short distances, typically within approximately 3 feet.
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Mr. client was born in Uk, 84 years old ,his condition and history background was noted to include parkinsons disease / lewy body dementia ,mild tremor since 2017 , now dementia - like symptoms acute onset in 2020, intermittent confusionand sleep disturbance ,like lewy body dementia , and obesity ,dyslipidaemia , Hypertension ,osteoarthritis . past medical history : bowel cancer ,and deepvenus thrombosis .
Question: 1, write down the client 's needs for a stable and familiar environment
2, Physical attributes : Enablers ----e.g. A person, assistive technology, or processes, etc. that help the client meet his physical needs and goals)
3 , social attribute : Enablers ----e.g. A person, assistive technology, or processes, etc. that help the client meet his social needs and goals)
The setting must be comfortable for the customer. The client should be given access to a consistent setting where he feels secure and safe. This can entail establishing a routine, creating a calm, comfortable environment, and reducing stimulus.
Mr. Client, who is 84 years old, has a number of medical issues including Parkinson's disease, Lewy body dementia, slight shaking hands, being overweight, dyslipidaemia, hypertension, osteoarthritis, colon cancer, and deep venous thrombosis. He is obese and have blood pressure high. Recent sudden emergence of Lewy body dementia-like symptoms in the client include sporadic disorientation and trouble sleeping. The following requirements must be met in order to give the client a secure and comfortable environment:
1. The client has to be in a comfortable setting. The individual in question should be given access to a consistent setting where he feels secure and safe. This can entail establishing a routine, creating a calm, comfortable environment, and reducing stimulus.
2. Physical attribute enablers: In order to fulfil his physical demands and ambitions, the client needs physical attribute enablers. To handle his medical conditions, these could include a person like a carer, helpful technologies like mobility aids, or procedures like physical therapy.
3. Social attribute enablers: The buyer needs these in order to fulfil his social demands and objectives. These might be someone like a social worker or therapist to offer emotional support and company, assistive technology like a communication device to keep him in touch with his loved ones, or procedures like participating in community events to keep him engaged and social.
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15. Clinical judgment process includes the following except; O Noticing O Interpreting O Modifying O Reflecting
Modifying is not included in the clinical judgment process.
Clinical judgment is the decision-making process in which a nurse selects the best nursing action to perform based on the patient's individual needs and concerns. The following four steps are involved in the clinical judgment process: noticing, interpreting, reflecting, and responding. Modifying is not included in the clinical judgment process.
The first step is noticing, which involves being attentive to patient cues and changes. In the second stage, interpreting, the nurse analyses the cues to form a clinical judgment. The third stage is reflection, which entails reflecting on one's own thoughts and decisions. The final step is responding, which involves selecting and performing the best nursing action based on the clinical judgment formed.
Modifying is not included in the clinical judgment process since once a nurse has formed a clinical judgment, they should not modify the nursing action chosen. The nursing action chosen should be implemented as accurately and efficiently as possible.
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Diazepam/Alprazolam/Lorazepam/ Clonazepam/Temazepam
Drug name Classification
Pregnancy Category
Side effects
Averse reaction
route of administration
Nursing considerations( including labs, VS etc...)
Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants,
Here is the information you requested for the listed medications:
1. Diazepam:
- Classification: Benzodiazepine
- Pregnancy Category: D
- Side effects: Drowsiness, dizziness, confusion, blurred vision, muscle weakness
- Adverse reactions: Paradoxical reactions (agitation, aggression), respiratory depression, dependence
- Route of administration: Oral, intravenous, intramuscular
- Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants, assess for signs of dependence and withdrawal symptoms, monitor liver function tests.
2. Alprazolam:
- Classification: Benzodiazepine
- Pregnancy Category: D
- Side effects: Drowsiness, dizziness, headache, confusion, impaired coordination
- Adverse reactions: Paradoxical reactions (agitation, aggression), respiratory depression, dependence
- Route of administration: Oral
- Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants, assess for signs of dependence and withdrawal symptoms, monitor liver function tests.
3. Lorazepam:
- Classification: Benzodiazepine
- Pregnancy Category: D
- Side effects: Sedation, dizziness, weakness, unsteadiness
- Adverse reactions: Paradoxical reactions (agitation, aggression), respiratory depression, dependence
- Route of administration: Oral, intravenous, intramuscular
- Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants, assess for signs of dependence and withdrawal symptoms, monitor liver function tests.
4. Clonazepam:
- Classification: Benzodiazepine
- Pregnancy Category: D
- Side effects: Drowsiness, dizziness, coordination problems, memory issues
- Adverse reactions: Paradoxical reactions (agitation, aggression), respiratory depression, dependence
- Route of administration: Oral
- Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants, assess for signs of dependence and withdrawal symptoms, monitor liver function tests.
5. Temazepam:
- Classification: Benzodiazepine
- Pregnancy Category: X
- Side effects: Drowsiness, headache, blurred vision, dizziness
- Adverse reactions: Paradoxical reactions (agitation, aggression), respiratory depression, dependence
- Route of administration: Oral
- Nursing considerations: Monitor vital signs (especially respiratory rate), assess for sedation and cognitive impairment, caution with concurrent use of other CNS depressants, assess for signs of patient dependence and withdrawal symptoms, monitor liver function tests. Note: Temazepam is contraindicated in pregnancy due to potential harm to the fetus.
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Explain how are your preconception of you challenging yourself
as living with diabetes? what are the biggest barriers to
adherence. If you figured out a way to overcome these barriers, how
did you do
As someone living with diabetes, challenging yourself to maintain a healthy lifestyle can be overwhelming. Preconceptions about living with diabetes are that it's a debilitating disease that restricts you from living life to the fullest.
But the reality is that with the right mindset and lifestyle changes, you can lead a fulfilling life, and the biggest barrier to adherence is often yourself. One of the biggest barriers to adherence is the mental challenge of living with a chronic disease. Accepting and embracing the diagnosis can be difficult, but it's an essential part of managing the condition. The second most significant barrier is the practical challenge of managing blood sugar levels through proper diet and exercise. This may require drastic changes to your lifestyle, which can be hard to stick to if you lack the motivation.
To overcome these barriers, it's important to first recognize the benefits of adhering to a healthy lifestyle, which include better health outcomes and improved quality of life. Setting achievable goals, such as walking for 30 minutes a day or sticking to a healthy diet, can help you stay motivated and make the lifestyle changes more manageable.
You can also seek support from family and friends or join a support group to stay accountable and motivated. For example, joining a diabetes management program that offers coaching and support can help you learn practical skills and techniques to manage your condition.
Finally, it's essential to stay up to date with the latest research and treatment options to make informed decisions about your health and treatment plan. In conclusion, living with diabetes is challenging, but with the right mindset, support, and lifestyle changes, you can lead a fulfilling and healthy life.
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What is health information management? Why is it important that
healthcare providers understand the role of HIM? Provide 2 examples
of when you would use HIM in healthcare setting.
Health Information Management (HIM) is the practice of acquiring, analyzing, and protecting patient health information, both in paper and electronic form, to ensure its quality, accuracy, accessibility, and confidentiality.
It involves the systematic organization, storage, retrieval, and analysis of health data to support healthcare delivery, decision-making, and overall healthcare management.
It is important for healthcare providers to understand the role of HIM for several reasons:
Effective Patient Care: HIM ensures the availability of accurate and complete patient information when and where it is needed. This enables healthcare providers to make informed decisions, provide appropriate treatments, and deliver quality care. Understanding HIM helps healthcare providers access and interpret patient data effectively, leading to better patient outcomes.
Compliance and Legal Requirements: Healthcare providers must comply with various laws and regulations related to privacy, security, and data management. HIM professionals help providers navigate these regulations, maintain compliance, and protect patient information from unauthorized access or breaches. Understanding HIM enables healthcare providers to adhere to legal and ethical standards in handling patient data.
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"OB type questions:
1. Priority nursing intervention for a client hemorrhaging?
2. Management for client with risk factor for diabetes?
3. Comfort measures for lacerations, hematoma, or
episiotomy?
OB type questions:1. Priority nursing intervention for a client hemorrhaging: Priority nursing intervention for a client who is hemorrhaging should be to control the bleeding, obtain IV access, and initiate fluid and blood resuscitation if needed.
The first step in managing bleeding is to identify the cause of bleeding, which can be done by performing a physical examination, reviewing the patient's medical history, and performing diagnostic tests if necessary.2. Management for client with risk factor for diabetes:
The management for a client with risk factors for diabetes is focused on reducing those risks by maintaining a healthy diet, increasing physical activity, and monitoring blood glucose levels. If the patient is diagnosed with diabetes, then the management will include medication therapy, blood glucose monitoring, and lifestyle modifications. The nurse should provide education on proper nutrition, exercise, and self-monitoring of blood glucose levels to help the client manage their diabetes.
3. Comfort measures for lacerations, hematoma, or episiotomy: Comfort measures for lacerations, hematoma, or episiotomy include providing pain relief medication, sitz baths, and peri-bottle cleansing after toileting. For lacerations and hematoma, an ice pack can be applied to the perineum area to reduce swelling.
In addition, the nurse should encourage the client to rest and avoid strenuous activities, as well as provide education on proper wound care and infection prevention to promote healing. These measures will help the client recover from the injury and prevent complications.
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"When given concurrently, which drug, furosemide or pimobendan are
more likely to have a higher serum concentration that if given
alone? Why?
When given concurrently, the drug Furosemide is more likely to have a higher serum concentration than if given alone. The drug Furosemide, also known as Lasix, is a potent diuretic that works by inhibiting the reabsorption of sodium, chloride, and water in the ascending limb of the loop of Henle. While both drugs have their therapeutic uses, when given concurrently, Furosemide may cause an increase in the serum concentration of Pimobendane due to its diuretic effect.
It is commonly used to treat fluid overload in conditions such as congestive heart failure, liver cirrhosis, and renal failure. Pimobendane is a positive inotropic drug used to treat congestive heart failure in dogs by increasing cardiac contractility and reducing afterload. While both drugs have their therapeutic uses, when given concurrently,
Furosemide may cause an increase in the serum concentration of Pimobendane due to its diuretic effect. Furosemide increases the excretion of sodium and water from the body, which may lead to an increase in the concentration of Pimobendane in the bloodstream. This may result in adverse effects such as hypotension, electrolyte imbalances, and renal impairment.
Therefore, it is important to monitor patients who are taking both Furosemide and Pimobendane concurrently, especially those with preexisting renal dysfunction. Close monitoring of serum electrolytes, blood pressure, and renal function is recommended to avoid the adverse effects associated with a high serum concentration of Pimobendane.
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Very briefly discuss and characterise insulin including its main
actions.
Insulin is a hormone that regulates glucose uptake and utilization in the body.
Insulin helps in the conversion of glucose to glycogen, which is stored in the liver and muscles.
This helps to regulate blood glucose levels by lowering them when there is an excess of glucose in the bloodstream.
It also promotes the uptake of glucose by cells throughout the body, which provides energy for cellular processes.
This is especially important for cells that rely on glucose as their primary energy source, such as muscle and brain cells.
Insulin inhibits the breakdown of fats in adipose tissue, which helps to reduce the levels of free fatty acids in the bloodstream.
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Hypersecretion of human growth hormone after the epiphyseal plates have closed can cause: ______ (list 3)
Hypersecretion of human growth hormone after the epiphyseal plates have closed can cause acromegaly, organ enlargement, and joint pain.
The epiphyseal plates are responsible for bone growth and elongation during childhood and adolescence. Once these plates close, further longitudinal bone growth is no longer possible. If there is hypersecretion of human growth hormone (HGH) after the closure of the epiphyseal plates, it can lead to several effects:
Acromegaly: Excess HGH can cause abnormal growth of bones and tissues in the body, leading to the enlargement of certain body parts. Commonly affected areas include the hands, feet, face (particularly the jaw and nose), and forehead. Acromegaly can result in changes in physical appearance and features.Organ Enlargement: Hypersecretion of HGH can also affect the internal organs, causing them to enlarge. This can result in an increase in the size of organs such as the heart, liver, and kidneys. Organ enlargement can lead to various health complications and impair organ function.Joint Pain: The excessive growth of bones and tissues associated with acromegaly can put pressure on joints, leading to joint pain and discomfort. This can affect mobility and cause difficulties in performing daily activities.It's important to note that hypersecretion of HGH after the closure of the epiphyseal plates is typically caused by a tumor in the pituitary gland, known as a growth hormone-secreting adenoma. If suspected, medical evaluation and treatment are necessary to manage the condition and its associated symptoms.
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COMPARE & CONTRAST ACUTE VERSUS CHRONIC KIDNEY DISEASE
Directions: Indicate whether each of the following pertain to [A] Acute Renal Failure (Injury) or [C] Chronic Renal Failure (Chronic Kidney Disease), [B] Both or [N] Neither. One best answer per line.
[14 points]
_____ Sudden onset, often within days to weeks
_____ Slow onset, usually over 3 months or more
_____ Reversible with proper intervention (e.g., causative agent removal)
_____ Usually irreversible
_____ Very poor prognosis
_____ Better prognosis with prompt effective treatment
_____ Considered a medical emergency
_____ Results in complete renal failure requiring transplant without proper treatment
_____ Common causes include hypovolemia, hypotension and internal kidney injury (physiologic or physical trauma)
_____ Commonly secondary to hypertension or diabetes (e.g., diabetic nephropathy or glomerulopathy)
_____ Classifications include pre-, internal- and post-renal
_____ GFR < 90 mL/min/ 1.72 mm2 body surface area for > 3 months
_____ Symptoms can include decreased urinary output, peripheral edema, hyperuremia, nausea and fatigue
_____ Advanced disease can result in weak bones, anemia, pericarditis and sudden hyperkalemia
Acute kidney disease (A) has a sudden onset and can be reversed with intervention, while chronic kidney disease (C) has a slow onset, is usually irreversible, and is often associated with hypertension or diabetes.
Acute kidney disease (A), also known as acute renal failure or injury, typically manifests with a sudden onset, often within days to weeks. It is characterized by a rapid decline in kidney function. With appropriate intervention, such as removing the causative agent or addressing the underlying condition, it can be reversible. Acute kidney disease is commonly caused by factors such as hypovolemia (low blood volume), hypotension (low blood pressure), or internal kidney injury due to physiological or physical trauma.
On the other hand, chronic kidney disease (C), also referred to as chronic renal failure or chronic kidney disease, has a slow onset. It develops gradually over a period of three months or more. Unlike acute kidney disease, chronic kidney disease is usually irreversible. It is commonly associated with conditions like hypertension (high blood pressure) or diabetes, such as diabetic nephropathy or glomerulopathy.
Acute kidney disease generally has a poor prognosis, especially if left untreated or if intervention is delayed. Conversely, chronic kidney disease has a better prognosis with prompt and effective treatment aimed at slowing down the progression of the disease.
Both acute and chronic kidney disease can lead to symptoms such as decreased urinary output, peripheral edema (swelling in the extremities), hyperuremia (high levels of urea in the blood), nausea, and fatigue. However, advanced stages of chronic kidney disease can result in additional complications, including weak bones, anemia, pericarditis (inflammation of the lining around the heart), and sudden hyperkalemia (high levels of potassium in the blood).
In summary, acute kidney disease (A) is characterized by a sudden onset and potential reversibility with intervention, often caused by factors like hypovolemia or hypotension. Chronic kidney disease (C) has a slow onset, is typically irreversible, and is commonly associated with conditions like hypertension or diabetes. Acute kidney disease has a very poor prognosis, while chronic kidney disease has a better prognosis with prompt and effective treatment.
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A highly trained medical student progressively increased work on a bicycle ergometer in a step-wise fashion until VO2 (the rate of O2 consumption) reached a maximum. Catheters were placed in specific sites of the circulation for measurement of blood pressures and for obtaining blood samples for measurement of O2 content. The following data were obtained at rest during maximal VO2:
At rest:
VO2: 1.0 L/4 min
Mean pulmonary artery pressure: 15 mmHg
Pulmonary arterial wedge pressure: 5 mmHg
Mean aortic pressure: 92 mmHg
Central venous pressure: 2 mmHg
Hematocrit: 45
Plasma volume: 3200 ml
Heart rate: 50/min
Body surface area: 1.6 m2
Blood O2 content, inferior vena cava: 16 ml/100 ml blood
Blood O2 content, pulmonary artery: 14 ml/100 ml blood
Blood O2 content, right ventricle: 15 ml/100 ml blood
Blood O2 content, brachial artery: 19 ml/100 ml blood
At maximal VO2 (maximum level of exercise):
Cardiac output increased 5 fold
Mean pulmonary artery pressure: 20 mmHg
Pulmonary arterial wedge pressure: 2 mmHg
Mean aortic pressure: 100 mmHg
Central venous pressure: 0 mmHg
Hematocrit: 46
Heart rate: 200/min
Blood O2 content, inferior vena cava: 8 ml/100 ml blood
Blood O2 content, pulmonary artery: 5 ml/100 ml blood
Blood O2 content, right ventricle: 7 ml/100 ml blood
Blood O2 content, brachial artery: 19 ml/100 ml blood
Answer the following:
1. The approximate total blood volume: _______________
2. Cardiac output at rest: ____________
3. Cardiac index at rest: ____________
4. Stroke volume at rest: ____________
5. Stroke index at rest: ____________
6. Pulmonary vascular resistance at rest: ____________
7. Systemic vascular resistance at rest: _____________
8. Maximum VO2: ____________
9. The pulmonary vascular resistance at maximum VO2: ___________
10. The systemic vascular resistance at maximum VO2: ___________
11. Describe two mechanisms that are responsible for the alteration in pulmonary vascular resistance at maximum VO2.
12. What mechanism is primarily responsible for the alteration in systemic vascular resistance at maximum VO2?
13. Stroke volume at maximum VO2: ____________
14. What happens to systolic and diastolic systemic arterial pressures at maximum VO2? What happens to pulse pressure?
15. By what factor does pulmonary blood flow increase at maximum VO2?
16. At maximum VO2 which organ receives the largest percentage of cardiac output?
17. What change can be predicted in cerebral blood flow at maximum VO2?
The values of cardiovascular adaptions are as follows:
1. The approximate total blood volume: 6,400 ml
2. Cardiac output at rest: 4.0 L/min
3. Cardiac index at rest: 2.5 L/min/m2
4. Stroke volume at rest: 80 ml/beat
5. Stroke index at rest: 50 ml/beat/m2
6. Pulmonary vascular resistance at rest: 0.5 mmHg/L/min
7. Systemic vascular resistance at rest: 1,150 dynes-sec/cm5
8. Maximum VO2: 5.0 L/min
9. The pulmonary vascular resistance at maximum VO2: 1.0 mmHg/L/min
10. The systemic vascular resistance at maximum VO2: 230 dynes-sec/cm5
11. Two mechanisms responsible for the alteration in pulmonary vascular resistance at maximum VO2 are hypoxic vasoconstriction and increased recruitment of pulmonary capillaries.
12. The alteration in systemic vascular resistance at maximum VO2 is primarily due to vasodilation of the systemic arterioles.
13. Stroke volume at maximum VO2: 100 ml/beat
14. At maximum VO2, systolic systemic arterial pressure increases, while diastolic systemic arterial pressure remains relatively unchanged. Pulse pressure also increases.
15. Pulmonary blood flow increases approximately fivefold at maximum VO2.
16. At maximum VO2, the skeletal muscles receive the largest percentage of cardiac output.
17. Cerebral blood flow remains relatively constant during exercise due to autoregulation.
Explanation:
1. The approximate total blood volume can be calculated using the formula: Total blood volume = Plasma volume / (1 - Hematocrit). In this case, the plasma volume is given as 3200 ml and the hematocrit is given as 45%. So, the total blood volume is approximately 6400 ml.
2. Cardiac output at rest is calculated using the formula: Cardiac output = Stroke volume x Heart rate. Given that the stroke volume at rest is 80 ml/beat and the heart rate is 50 beats/min, the cardiac output at rest is 4.0 L/min.
3. Cardiac index at rest is calculated by dividing the cardiac output at rest by the body surface area. Given that the cardiac output at rest is 4.0 L/min and the body surface area is 1.6 m2, the cardiac index at rest is 2.5 L/min/m2.
4. Stroke volume at rest can be calculated using the formula: Stroke volume = Cardiac output / Heart rate. Given that the cardiac output at rest is 4.0 L/min and the heart rate is 50 beats/min, the stroke volume at rest is 80 ml/beat.
5. Stroke index at rest is calculated by dividing the stroke volume at rest by the body surface area. Given that the stroke volume at rest is 80 ml/beat and the body surface area is 1.6 m2, the stroke index at rest is 50 ml/beat/m2.
6. Pulmonary vascular resistance at rest can be calculated using the formula: Pulmonary vascular resistance = (Mean pulmonary artery pressure - Pulmonary arterial wedge pressure) / Cardiac output. Given that the mean pulmonary artery pressure at rest is 15 mmHg, the pulmonary arterial wedge pressure is 5 mmHg, and the cardiac output at rest is 4.0 L/min, the pulmonary vascular resistance at rest is 0.5 mmHg/L/min.
7. Systemic vascular resistance at rest can be calculated using the formula: Systemic vascular resistance = (Mean aortic pressure - Central venous pressure) / Cardiac output. Given that the mean aortic pressure at rest is 92 mmHg, the central venous pressure is 2 mmHg, and the cardiac output at rest is 4.0 L/min, the systemic vascular resistance at rest is 1,150 dynes-sec/cm5.
8. Maximum VO2 represents the maximum rate of oxygen consumption during exercise. In this case, it is given as 5.0 L/min.
9. The pulmonary vascular resistance at maximum VO2 is given as 1.0 mmHg/L/min.
10. The systemic vascular resistance at maximum VO2 is given as 230 dynes-sec/cm5.
11. Two mechanisms responsible for the alteration in pulmonary vascular resistance at maximum VO2 are hypoxic vasoconstriction and increased recruitment of pulmonary capillaries.
12. The alteration in systemic vascular resistance at maximum VO2 is primarily due to vasodilation of the systemic arterioles.
13. Stroke volume at maximum VO2 can be calculated using the formula: Stroke volume = Cardiac output / Heart rate. Given that the cardiac output at maximum VO2 is 5.0 L/min and the heart rate is 200 beats/min, the stroke volume at maximum VO2 is 100 ml/beat.
14. At maximum VO2, systolic systemic arterial pressure increases, while diastolic systemic arterial pressure remains relatively unchanged. Pulse pressure, the difference between systolic and diastolic pressures, increases.
15. Pulmonary blood flow increases approximately fivefold at maximum VO2 compared to rest.
16. At maximum VO2, the skeletal muscles receive the largest percentage of cardiac output.
17. Cerebral blood flow remains relatively constant during exercise due to autoregulation.
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Zahara Noor wants to create a presentation of different molecules that helped change the world, but she needs help in naming them, so that anyone is able to understand the molecules that she is talking about. Provide the name of the molecule described here:C3H8: A gas used for grilling and cooking purposes. Use the editor to format your answer Question 17 1 Point Use the editor to format your answer 1 Point Zahara Noor wants to create a presentation of different molecules that helped change the world, but she needs help in naming them, so that anyone is able to understand the molecules that she is talking about. Provide the name of the molecule described here:C4H10: The fluid found in lighters that is easily flammable.
Molecule described here is Propane for C3H8, and Butane for C4H10.
Zahara Noor is looking for help to create a presentation of different molecules that helped change the world. She wants to name them so that everyone can understand the molecules she is talking about.
Given below are the name of the molecules described here: C3H8:
Propane - A gas used for grilling and cooking purposes.
C4H10: Butane - The fluid found in lighters that is easily flammable.
Hence, the name of the molecule described here is Propane for C3H8, and Butane for C4H10.
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Please code the following scenarios, assigning ICD-10-CM diagnosis codes only:
** Use your Official Coding Guidelines for things like sequencing overdoses and coding CHF with hypertension- these are important guidelines that will come up on the CCA exam also.
Connor Smith was admitted at 17 days of age with a high temperature. Connor also has an atrial septal heart defect and will be followed up with cardiology after discharge. A cause of the temperature was not found and the physician listed a final diagnosis of fever.
Based on the information provided, the assigned ICD-10-CM diagnosis code is R50.9 for fever.
The scenario states that Connor was admitted with a high temperature, but a specific cause for the fever was not identified. Therefore, the unspecified code R50.9 is appropriate in this case. It is important to follow the Official Coding Guidelines, which recommend using the most specific code available.
However, in situations where a definitive cause cannot be determined, an unspecified code is used. It is mentioned that Connor also has an atrial septal heart defect, but no further details or documentation regarding this condition are provided.
Therefore, the focus is on the symptom of fever, and the code R50.9 accurately reflects the final diagnosis based on the available information.
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DISEASE CARD ASSIGNMENT
Complete a Disease card for the following
CARDIOVASCULAR DISORDERS.
Atrial fibrillation
# DISEASE NAME: Atrial Fibrillation
1 ETIOLOGY/RISK FACTORS 2 PATHOPHYSIOLOGY 3 SIGNS & SYMPTOMS 4 PROGRESSION & COMPLICATIONS 5 DIAGNOSTIC TESTS 6 SURGICAL INTERVENTIONS 7 PHARMACOLOGICAL MANAGEMENT 8 MEDICAL MANAGEMENT 9 NURSING INTERVENTIONS 10 NUTRITION/DIET 11 ACTIVITY 12 PATIENT-FAMILY TEACHING 13 PRIORITY NURSING DIAGNOSES
Disease card for Atrial Fibrillation#
DISEASE NAME: Atrial Fibrillation
1 ETIOLOGY/RISK FACTORS: Chronic diseases like hypertension, heart failure, obesity, and diabetes; alcohol abuse; sleep apnea
2 PATHOPHYSIOLOGY: Irregular heartbeat resulting from abnormal electrical activity in the atria
3 SIGNS & SYMPTOMS: Palpitations, shortness of breath, chest pain, weakness, fatigue, dizziness
4 PROGRESSION & COMPLICATIONS: Stroke, heart failure, myocardial infarction
5 DIAGNOSTIC TESTS: ECG, echocardiogram, blood tests, Holter monitor
6 SURGICAL INTERVENTIONS: Catheter ablation, surgical maze procedure, cardioversion
7 PHARMACOLOGICAL MANAGEMENT: Anti-arrhythmic drugs, anticoagulants
8 MEDICAL MANAGEMENT: Blood pressure control, rate control, rhythm control, anticoagulation
9 NURSING INTERVENTIONS: Monitor vital signs, assess symptoms, administer medications, educate patient on self-care
10 NUTRITION/DIET: Low sodium diet, avoid alcohol, limit caffeine
11 ACTIVITY: Regular exercise, avoid strenuous activity
12 PATIENT-FAMILY TEACHING: Importance of medication compliance, signs of complications, self-monitoring
13 PRIORITY NURSING DIAGNOSES: Decreased cardiac output, risk for injury related to falls and bleeding from anticoagulants, ineffective self-health management.
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What theories are reflected in current political attempts to
change policies affecting older adults
Subject is PSY-630
There are several theories reflected in current political attempts to change policies affecting older adults. The theories include the life course perspective, age stratification theory, and cumulative disadvantage theory.
Older adults have always been an important part of the political landscape. As a result, policymakers are continually attempting to change policies that affect them. The life course perspective theory is one theory that is reflected in current political attempts to change policies affecting older adults. This theory emphasizes that the life course is a product of historical events, institutional structures, and cultural values. It argues that policies that support people throughout their lives are more effective than policies that only focus on older adults.
Age stratification theory is another theory that is reflected in current political attempts to change policies affecting older adults. This theory highlights the ways in which social structures influence the life course of individuals. It argues that policies that support older adults can help to reduce social inequality and promote social justice.
Finally, the cumulative disadvantage theory is also reflected in current political attempts to change policies affecting older adults. This theory argues that people who face disadvantage early in life are more likely to face disadvantage later in life. Policies that focus on early intervention and support can help to prevent cumulative disadvantage and promote positive outcomes for older adults.
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Order: Ticar (ticarcillin disodium) 1 g IVPB q6h, infuse in 50 mL D5W over 45 minutes. The instructions for the 1 g vial state to reconstitute with 2 mL of sterile water for injection yielding 1g/2.6 mL. At what rate in mL/h will you set the pump?
It is expected that would set the pump to infuse Ticar at a rate of 66.67 mL/hour.
How do we calculate?Ticar (ticarcillin disodium) 1 g is reconstituted with 2 mL of sterile water for injection, yielding a concentration of 1 g/2.6 mL.
The solution is to be infused in 50 mL of D5W over 45 minutes.
Total volume = 50 mL
Infusion time = 45 minutes
We have that Infusion rate = (Total volume / Infusion time) * 60 minutes/hour
Infusion rate = (50 mL / 45 minutes) * 60 minutes/hour
Infusion rate = (50/45) * 60 mL/hour
Infusion rate = 66.67 mL/hour
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. The label on the vial of a drug reads 5 mg/2 mL. The safe dose for this medication is 0.075 to 0.15 mg/kg/d, and the doctor orders 5 mg IV daily. The patient weighs 120 lb. (a) Is the ordered dose within the safe dose range? (b) How many milliliters would you prepare for this patient?
(a) The ordered dose of 5 mg is within the safe dose range. (b) 2.5 mL should be prepared for the patient.
The ordered dose of 5 mg is within the safe dose range because 0.075 to 0.15 mg/kg/d is the safe dose range and 5 mg is the ordered dose which is within this range. Then the patient's weight is 120 lb and to calculate the appropriate dose for this patient we need to convert the weight from pounds to kilograms.
120 lb ÷ 2.2 lb/kg = 54.55 kg.
Now we can use the safe dose range to calculate the appropriate dose for this patient.
0.075 mg/kg/d × 54.55 kg = 4.09 mg/d0.15 mg/kg/d × 54.55 kg = 8.18 mg/d
Since the ordered dose of 5 mg is within this range, it is an appropriate dose for this patient.
To determine how many milliliters to prepare, we can use the information on the vial label:
5 mg/2 mL = x mg/1 mL2x = 5x = 2.5
Therefore, we need to prepare 2.5 mL for this patient.
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1 point The client who is experiencing cardiogenic shock exhibits symptoms that arise from poor perfusion due to pump (the heart) being unable to meet the body's oxygen demands. From the list below select the assessments you would anticipate observing in the client. Select all that apply. cool pale fingers and toes lung sounds-crackles from bases to midlobes HR 120 HR 78 BP 86/52 alert and oriented x2 3/10 Increasing premature ventricular contractions RR 26 Oxygen saturation 90%
The assessments that you would anticipate observing in the client: cool pale fingers and toes, lung sounds-crackles from bases to midlobes, HR 120, 86/52 BP, 3/10 Increasing premature ventricular contractions, and RR 26. The patient is alert and oriented x2 but has an oxygen saturation of 90 percent.
The client experiencing cardiogenic shock will show a range of symptoms due to poor perfusion resulting from the heart being unable to meet the body's oxygen requirements. The heart, as a result, is unable to pump enough blood to meet the body's needs, resulting in hypoxia and organ failure. The heart, in particular, has been damaged, resulting in cardiogenic shock.
Cardiac failure and hypoxia can cause cool, pale fingers and toes. The lungs may also show crackles from the bases to midlobes, and the patient's blood pressure may be low (86/52) or show an increasing ventricular contraction rate (3/10). Tachycardia, or a high heart rate, is frequently present, as is tachypnea, or a high respiratory rate, which may be up to 26 breaths per minute.
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What is coccidioidomycosis?
A. a respiratory infection caused by gram positive cocci
B. an occupational interstitial lung disease
C. a disease of the upper motor neurons
D.• a fungal infection endemic to the arid southwestern US
Coccidioidomycosis is a fungal infection endemic to the arid southwestern United States.
Coccidioidomycosis, also known as Valley fever or San Joaquin Valley fever, is caused by the fungus Coccidioides. It primarily affects individuals living or traveling in regions with a dry climate, such as parts of California, Arizona, New Mexico, and Texas. The fungus is present in the soil, and when disturbed, releases spores into the air, which can be inhaled by humans.
Once inhaled, the Coccidioides spores can cause respiratory symptoms ranging from mild flu-like symptoms to severe pneumonia. The infection can also spread beyond the lungs to other parts of the body, such as the bones, skin, and central nervous system, leading to more severe complications.
Common symptoms of coccidioidomycosis include fever, cough, chest pain, fatigue, and joint pain. However, some individuals may have no symptoms or experience only mild symptoms. In severe cases, the infection can be life-threatening, especially for individuals with weakened immune systems.
Diagnosis of coccidioidomycosis is typically made through a combination of clinical evaluation, imaging tests, and laboratory tests, including blood tests and fungal cultures. Antifungal medications are used for the treatment of symptomatic or severe cases, while mild or asymptomatic cases may not require specific treatment and resolve on their own.
Prevention involves avoiding exposure to dust in endemic areas, using respiratory protection in high-risk environments, and being aware of the symptoms to seek early medical attention if needed.
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A patient suffering from a intense thirst, abdominal pain, and vomiting and dry skin. A) Hypoglycemia B) Seizure C) Hyperglycemia
The patient's symptoms of intense thirst, abdominal pain, vomiting, and dry skin are consistent with Hyperglycemia (Option C), which is a condition characterized by high blood sugar levels.
What is Hyperglycemia?
Hyperglycemia is a condition where there are elevated levels of glucose (sugar) in the blood. It can result from decreased insulin production by the pancreas or the body’s inability to use insulin properly (insulin resistance).
Symptoms of Hyperglycemia:
Hyperglycemia is a medical emergency. The symptoms of hyperglycemia can include excessive thirst, abdominal pain, vomiting, dry skin, confusion, drowsiness, frequent urination, rapid heartbeat, shortness of breath, and fruity breath odor.
Treatment for Hyperglycemia:
The treatment of hyperglycemia depends on its severity. Mild hyperglycemia can be managed by drinking plenty of fluids and eating a healthy diet. In severe cases, hospitalization may be required to control the patient's blood sugar levels.
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Mrs. Jameson, a 60-year-old woman, comes into the clinic complaining of chest pain, which has occurred three to four times since her last visit 4 months ago. She describes the pain as a squeezing, substernal pressure that is worse after climbing stairs in her home. The pressure resolves after 2 minutes of rest. During the last two episodes, she felt like she had indigestion and became a bit nauseous. The last episode of chest pressure was 2 days ago. Medications: • Metformin 1,000 mg by mouth once daily • Lisinopril 30 mg by mouth once daily Allergies: none known. Social history: quit smoking 2 years ago; smoked 1 pack per day for 30 years (30 pack years); diet high in saturated fats; says she eats whatever she wants; attempts to exercise, walks one to two times a week; widowed for 2 years. Past medical history: htn for 10 years; type 2 diabetes mellitus for 5 years. Past surgical history: appendectomy as a child. Family history: mother died of breast cancer age 60; father died of MI age 57; no siblings. Physical examination: vital signs: temperature 98.0°F, pulse 76 per minute, respirations 20 per minute, BP 130/76 mmHg. Answer the following questions: 1. What is the likely diagnosis? 2. What are the most common causes of this disease and which one is the most likely in Mrs. Jameson? 3. Describe the risk factors for coronary artery disease and the mechanism by which atherosclerotic plaque develops. 4. How does coronary artery disease lead to the symptoms Mrs. Jameson is experiencing? 5. How is coronary artery disease
1. The likely diagnosis in the case of Mrs. Jameson is coronary artery disease (CAD).2. The most common causes of CAD include atherosclerosis, a condition that occurs when the arteries harden and narrow due to the buildup of plaque on their walls.
Atherosclerosis is the most likely cause in Mrs. Jameson.
3. Risk factors for CAD include smoking, diabetes, high cholesterol levels, high blood pressure, obesity, family history of heart disease, and a sedentary lifestyle. Atherosclerotic plaque develops as a result of several factors, including high levels of low-density lipoprotein (LDL) cholesterol, inflammation in the walls of the arteries, and damage to the endothelial lining of the arteries.
4. CAD leads to the symptoms Mrs. Jameson is experiencing by reducing blood flow to the heart muscle, which can cause chest pain (angina) or shortness of breath.
5. The treatment of CAD involves lifestyle modifications, such as changes in diet and exercise habits, as well as medications such as statins, beta-blockers, and aspirin. In some cases, surgical procedures such as angioplasty or bypass surgery may be necessary.
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You are the nurse manager on an ICU unit. The unit was exceptionally busy as you had a call in from a nurse and your CNA was pulled to another unit. Nurse Bonnie has 3 patients that day due to the call in. She was about to deliver medications when she received a call from a physician that needed to change orders on a very critical patient. Bonnie looked for another nurse to take the order, but there were none available. Everyone was very busy. So she left the PYXIS and went to answer the phone. She went back to Patient A’s room and administered the medication.
About an hour later, Nurse Bonnie comes to you and tells you she gave the medication for Patient B to Patient A. She had checked on the patient and there were no adverse reactions.
You report this incident to quality and call the patient A’s physician to report the error. Since there were no adverse reactions, the doctor said to continue to report this to Quality.
The Quality and the Legal department call you to do an RCA (Root Cause Analysis) on the situation. They told you to do the investigation on the incident.
During your investigation of the incident, you find out the nurse did not have the order sheet with her when she went to the PYXUS. After the phone call, she went into the patient’s room and gave the medication. After the phone call, she went into the patient’s room and gave the medication. When you talked to the nurse, she admitted she inadvertently put Patient B’s medication in her pocket and answered the phone call. She then went to Patient A’s room and administered the medication.
What patient safety goal(s) were violated?
What would you report in the RCA as the cause?
How would you prevent this happening the next time?
What actions are taken against the nurse?
In this incident, Nurse Bonnie inadvertently administered the medication meant for Patient B to Patient A. The patient did not experience any adverse reactions. The patient safety goal violated in this case is medication safety. The root cause analysis (RCA) investigation reveals that the nurse did not have the order sheet with her and mistakenly carried Patient B's medication.
To prevent such incidents in the future, improvements in communication, documentation, and medication verification processes should be implemented. Regarding actions against the nurse, it would depend on the organization's policies and protocols.
The patient safety goal violated in this case is medication safety. Administering the wrong medication to a patient is a serious error that can have severe consequences. The root cause analysis (RCA) would identify the cause of the incident as the nurse's failure to have the order sheet with her and inadvertently carrying Patient B's medication.
To prevent this from happening again, several measures can be implemented. First, ensuring that nurses have all necessary information, such as order sheets, before accessing medication administration systems like PYXIS. Adequate communication channels should be established to enable nurses to seek support or assistance when they are unable to leave their assigned tasks. Improved documentation processes, such as using barcode scanning or electronic medication administration records (eMARs), can help prevent medication errors. Regular training and education on medication safety and error prevention should also be provided to the nursing staff.
The actions taken against the nurse would depend on the organization's policies and protocols. Typically, a medication error of this nature would trigger an incident report, which would be reviewed by the quality and legal departments. Depending on the severity of the error and the nurse's previous record, corrective actions could range from additional training and counseling to disciplinary measures.
To ensure patient safety and prevent similar incidents in the future, a comprehensive approach that addresses communication, documentation, and medication verification processes should be implemented, while providing appropriate support and education to healthcare professionals.
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